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p erk  (Cell Signaling Technology Inc)


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    Structured Review

    Cell Signaling Technology Inc p erk
    P Erk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 13438 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/p+erk/Phospho-p44%2F42+MAPK+(Erk1%2F2)+(Thr202%2FTyr204)+XP+Rabbit+mAb/pmc13042271-96-58-63
    Average 99 stars, based on 13438 article reviews
    p erk - by Bioz Stars, 2026-10
    99/100 stars

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    other:

    Article Title: Novel Hsp90 inhibitor JD‑02 inhibits HSV‑1 infection via the Raf/MEK/ERK signaling pathway.
    Article Snippet: These primary antibodies included IcP0 (1:1,000; cat. no. ab6513; Abcam), IcP27 (1:1,000; cat. no. ab53480; Abcam), gd (1:1,000; cat. no. ab6507; Abcam), gB (1:500; cat. no. sc‐56987; Santa cruz Biotechnology, Inc.), Raf‐B (1:500; cat. no. sc‐166; Santa cruz Biotechnology, Inc.), p‐BRAF (1:1,000; cat. no. 2696T; cell Signaling Technology, Inc.), ERK (1:1,000; cat. no. 4695S; cell Signaling Technology, Inc.), p‐ERK (1:1,000; cat. no. 4370S; cell Signaling Technology, Inc.), MEK1/2 (1:1,000; cat. no. AF6385; Affinity Biosciences), p‐MEK1/2 (1:1,000; cat. no. 9154T; cell Signaling Technology, Inc.), Flag (1:1,000; cat. no. 14793S; cell Signaling Technology, Inc.), HA (1:1,000; cat. no. 3724S; cell Signaling Technology, Inc.).

    Article Title: Novel Hsp90 inhibitor JD-02 inhibits HSV-1 infection via the Raf/MEK/ERK signaling pathway
    Article Snippet: These primary antibodies included ICP0 (1:1,000; cat. no. ab6513; Abcam), ICP27 (1:1,000; cat. no. ab53480; Abcam), gD (1:1,000; cat. no. ab6507; Abcam), gB (1:500; cat. no. sc-56987; Santa Cruz Biotechnology, Inc.), Raf-B (1:500; cat. no. sc-166; Santa Cruz Biotechnology, Inc.), p-BRAF (1:1,000; cat. no. 2696T; Cell Signaling Technology, Inc.), ERK (1:1,000; cat. no. 4695S; Cell Signaling Technology, Inc.), p-ERK (1:1,000; cat. no. 4370S; Cell Signaling Technology, Inc.), MEK1/2 (1:1,000; cat. no. AF6385; Affinity Biosciences), p-MEK1/2 (1:1,000; cat. no. 9154T; Cell Signaling Technology, Inc.), Flag (1:1,000; cat. no. 14793S; Cell Signaling Technology, Inc.), HA (1:1,000; cat. no. 3724S; Cell Signaling Technology, Inc.).

    Article Title: Rhamnogalacturonan rich fractions isolated from Saururus chinensis alleviate ulcerative colitis via regulation of MAPK/NF-κB pathway and tight junction
    Article Snippet: The primary antibodies for p-ERK, p-p38, and inhibitor nuclear factor-kappa B-alpha (IκBα) were purchased from Cell Signaling Technology (Danvers, MA, USA).

    Article Title: Adipose stem cells-derived microvesicles and chicken egg-derived exosomes attenuate cardiac ischemia/reperfusion injury through AKT/ERK/Nrf2/HO-1 axis to inhibit apoptosis and inflammation and restore autophagy.
    Article Snippet: Aims: Myocardial ischemia/reperfusion (I/R) injury is an unsolved medical issue that is caused by additional injuries derived from reperfusion therapy for patients with acute myocardial infarction, one of the leading causes of morbidity and mortality in the world.. Myocardial I/R injury causes unpredictable complications evoked by oxidative stress, endothelial dysfunction, dysregulated autophagy, apoptosis, and an imbalanced inflammatory response.. Microvesicles (MVs) derived from adipose stem cells (ADSC) and egg-derived exosomes (EXOs) may confer antioxidant, anti-inflammatory, anti-apoptotic and tissue repair Jo ur na l P re -p ro of Journal Pre-proof potential, showing potential in cardiovascular therapy.

    Article Title: SKA2 promotes gastric cancer progression by regulating glutathione metabolism
    Article Snippet: p-ERK , Cell Signaling Technology , Cat#4370; RRID: AB_2315112.

    Article Title: Antibodies against IGFR-like receptor and uses thereof
    Article Snippet: The following antibodies were used with corresponding dilutions: IGFR-like receptor SEQ ID No. 2 (1:5000 rabbit Pineda, Berlin, Germany), IGFR-like receptor SEQ ID No. 2 (1:100 rat and mouse), IGFR-like receptor SEQ ID No. 4 and 6 (1:10 rat and mouse), Akt (1:5000 Cell Signaling, 4691), P-Akt (1:5000 Cell Signaling, 4060), Erk (1:5000 Cell Signaling, 4695), P-Erk (1:5000 Cell Signaling, 4370), m-Tor (1:1000 Cell Signaling, 2972), P-mTor (1:1000 Cell Signaling, 5536), Ampk (1:1000 Cell Signaling, 2532), P-Ampk (1:2500 Cell Signaling, 2535), IRS-2 (1:1000 Cell Signaling, 4502), IRβ (1:1000 Cell Signaling, 3025), S6rp (1:5000 Cell Signaling, 2217S), P-S6RP (1:5000 Cell Signaling, 2211S), P-IR/IGFR (1:1000, Millipore, 07-841), α-Tubulin (1:5000, Sigma, T7451), mouse Adaptin beta clone 74 (1:5000, BD Bioscience 610382).

    Incubation:

    Article Title: Curcumin protects retinal photoreceptors in Glaucoma by activating prostaglandin D2 synthase-dependent NRF2 signaling.
    Article Snippet: This is a PDF of an article that has undergone enhancements after acceptance, such as the addition of a cover page and metadata, and formatting for readability.. This version will undergo additional copyediting, typesetting and review before it is published in its final form.. As such, this version is no longer the Accepted Manuscript, but it is not yet the definitive Version of Record; we are providing this early version to give early visibility of the article.

    Article Title: Single-stranded DNA in the bone microenvironment promotes prostate cancer bone metastasis via the ITGA6-FAK pathway.
    Article Snippet: .. Blots were incubated with primary antibodies described as the following: FAK (Forevertech Biotechnologies Co., Ltd, 20431, 1:1000), p-FAK (Tyr397) (CST, 3283S, 1:1000), AKT (CST, 9272S, 1:1000), p-AKT (CST, 4060S, 1:1000), ERK (CTS, 9102S, 1:1000), p-ERK (CST, 4370S, 1:1000), ITGA6 (proteintech, 27189-1-AP, 1:1000), ITGB4 (proteintech, 21738-1-AP, 1:1000), and GAPDH (Origene, TA802519, 1:5000). .. Secondary antibodies including Goat anti-Mouse IgG (H+L) Secondary Antibody, HRP (Invitrogen, AB_228307) and Goat anti-Rabbit IgG (H+L) Secondary Antibody, HRP (Invitrogen, AB-228341) were subsequently applied.



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    GA effects <t>on</t> <t>ERK1/2</t> phosphorylation in invasive ductal carcinoma-derived KAIMRC1 stem-like cells. Representative western blot analyses showed p-ERK1/2 in KAIMRC1 stem-like cells treated with 100 µg/ml GA for (A) 30 to 120 min and with (B) 50–100 µg/ml GA or unglycated BSA for 90 min of incubation. Bar graphs show the relative expression level of p-ERK1/2 normalized to total ERK1/2. GAPDH served as the loading control. Data are presented as the mean ± SD from three independent experiments. Statistical significance relative to the untreated control is indicated as *P < 0.05 and **P < 0.01. C, control; GA, glycated albumin; p-ERK1/2, phosphorylated-ERK1/2; GAPDH, glyceraldehyde 3-phosphate dehydrogenase.
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    GA effects <t>on</t> <t>ERK1/2</t> phosphorylation in invasive ductal carcinoma-derived KAIMRC1 stem-like cells. Representative western blot analyses showed p-ERK1/2 in KAIMRC1 stem-like cells treated with 100 µg/ml GA for (A) 30 to 120 min and with (B) 50–100 µg/ml GA or unglycated BSA for 90 min of incubation. Bar graphs show the relative expression level of p-ERK1/2 normalized to total ERK1/2. GAPDH served as the loading control. Data are presented as the mean ± SD from three independent experiments. Statistical significance relative to the untreated control is indicated as *P < 0.05 and **P < 0.01. C, control; GA, glycated albumin; p-ERK1/2, phosphorylated-ERK1/2; GAPDH, glyceraldehyde 3-phosphate dehydrogenase.
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    Image Search Results


    GA effects on ERK1/2 phosphorylation in invasive ductal carcinoma-derived KAIMRC1 stem-like cells. Representative western blot analyses showed p-ERK1/2 in KAIMRC1 stem-like cells treated with 100 µg/ml GA for (A) 30 to 120 min and with (B) 50–100 µg/ml GA or unglycated BSA for 90 min of incubation. Bar graphs show the relative expression level of p-ERK1/2 normalized to total ERK1/2. GAPDH served as the loading control. Data are presented as the mean ± SD from three independent experiments. Statistical significance relative to the untreated control is indicated as *P < 0.05 and **P < 0.01. C, control; GA, glycated albumin; p-ERK1/2, phosphorylated-ERK1/2; GAPDH, glyceraldehyde 3-phosphate dehydrogenase.

    Journal: Oncology Letters

    Article Title: Methylglyoxal-derived glycated albumin enhances the stemness potential of invasive ductal carcinoma-derived breast cancer stem-like cell line KAIMRC1

    doi: 10.3892/ol.2026.15541

    Figure Lengend Snippet: GA effects on ERK1/2 phosphorylation in invasive ductal carcinoma-derived KAIMRC1 stem-like cells. Representative western blot analyses showed p-ERK1/2 in KAIMRC1 stem-like cells treated with 100 µg/ml GA for (A) 30 to 120 min and with (B) 50–100 µg/ml GA or unglycated BSA for 90 min of incubation. Bar graphs show the relative expression level of p-ERK1/2 normalized to total ERK1/2. GAPDH served as the loading control. Data are presented as the mean ± SD from three independent experiments. Statistical significance relative to the untreated control is indicated as *P < 0.05 and **P < 0.01. C, control; GA, glycated albumin; p-ERK1/2, phosphorylated-ERK1/2; GAPDH, glyceraldehyde 3-phosphate dehydrogenase.

    Article Snippet: Mouse primary monoclonal antibodies directed against extracellular signal-regulated kinase (ERK)1 (clone G-8; cat. no. sc-271269), phosphorylated-ERK1/2 (p-ERK1/2; clone E-4; Tyr204 of ERK1; cat. no. sc-7383), OCT3/4 (clone A-9; cat. no. sc-365509) and RAGE (clone E-1; cat. no. sc-74473) were obtained from Santa Cruz Biotechnology, Inc.

    Techniques: Phospho-proteomics, Derivative Assay, Western Blot, Incubation, Expressing, Control